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Use este identificador para citar ou linkar para este item: https://ri.ufs.br/jspui/handle/riufs/23842
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dc.contributor.authorMorvan, Eduardo-
dc.contributor.authorLima, Nathalia Edviges Alves-
dc.contributor.authorMachi, Jacqueline Freire-
dc.contributor.authorMostarda, Cristiano-
dc.contributor.authorDe Angelis, Kátia-
dc.contributor.authorIrigoyen, Maria Cláudia-
dc.contributor.authorWichi, Rogério Brandão-
dc.contributor.authorRodrigues, Bruno-
dc.contributor.authorMaifrino, Laura Beatriz Mesiano-
dc.date.accessioned2025-11-12T19:15:46Z-
dc.date.available2025-11-12T19:15:46Z-
dc.date.issued2013-
dc.identifier.citationMORVAN, E. et al. Metabolic, hemodynamic and structural adjustments to low intensity exercise training in a metabolic syndrome model. Cardiovascular Diabetology, London, v. 12, n. 89, 2013. Disponível em: https://cardiab.biomedcentral.com/articles/10.1186/1475-2840-12-89. Acesso em: 12 nov. 2025.pt_BR
dc.identifier.issn1475-2840-
dc.identifier.urihttps://ri.ufs.br/jspui/handle/riufs/23842-
dc.languageengpt_BR
dc.publisherBioMed Centralpt_BR
dc.relation.ispartofCardiovascular Diabetologypt_BR
dc.subjectFructoseeng
dc.subjectExercise trainingeng
dc.subjectTriglycerideseng
dc.subjectInsulin resistanceeng
dc.subjectBaroreflex sensitivityeng
dc.subjectCardiovascular remodelingeng
dc.subjectAorta remodelingeng
dc.titleMetabolic, hemodynamic and structural adjustments to low intensity exercise training in a metabolic syndrome modelpt_BR
dc.typeArtigopt_BR
dc.identifier.licenseCreative Commons Atribuição 2.0 Genérica (CC BY 2.0)pt_BR
dc.description.resumoBackground: The increase in fructose consumption is paralleled by a higher incidence of metabolic syndrome, and consequently, cardiovascular disease mortality. We examined the effects of 8 weeks of low intensity exercise training (LET) on metabolic, hemodynamic, ventricular and vascular morphological changes induced by fructose drinking in male rats. Methods: Male Wistar rats were divided into (n = 8 each) control (C), sedentary fructose (F) and ET fructose (FT) groups. Fructose-drinking rats received D-fructose (100 g/l). FT rats were assigned to a treadmill training protocol at low intensity (30% of maximal running speed) during 1 h/day, 5 days/week for 8 weeks. Measurements of triglyceride concentrations, white adipose tissue (WAT) and glycemia were carried out together with insulin tolerance test to evaluate metabolic profile. Arterial pressure (AP) signals were directly recorded. Baroreflex sensitivity (BS) was evaluated by the tachycardic and bradycardic responses. Right atria, left ventricle (LV) and ascending aorta were prepared to morphoquantitative analysis. Results: LET reduced WAT (−37.7%), triglyceride levels (−33%), systolic AP (−6%), heart weight/body weight (−20.5%), LV (−36%) and aortic (−76%) collagen fibers, aortic intima-media thickness and circumferential wall tension in FT when compared to F rats. Additionally, FT group presented improve of BS, numerical density of atrial natriuretic peptide granules (+42%) and LV capillaries (+25%), as well as the number of elastic lamellae in aorta compared with F group. Conclusions: Our data suggest that LET, a widely recommended practice, seems to be particularly effective for preventing metabolic, hemodynamic and morphological disorders triggered by MS.pt_BR
dc.description.localLondonpt_BR
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